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ZfuerKardiologie.80870015.eng.abstr | ZfuerKardiologie.80870015.eng.abstr | [
{
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"Cheyne-Stokes respiration ( CSR ) during sleep is common in patients with severe congestive heart failure , induces repetitive oxygen desaturation with arousals , and impairs sleep . This causes daytime symptoms and likely an increase in sympathetic activity . It has , therefore , been suggested that CSR is independently related to mortality . The major mechanisms behind CSR induce reduced body stores of oxygen , a low apneic threshold for carbon dioxide , prolonged circulation time between the lung and the carotid body , and distrubance of respiratory control due to arousals . It is apparent that the main task in treating CSR is the therapy of congestive heart failure . Indeed , diuretics to treat pulmonary congestion as well as ACE-inhibitors reduce CSR . Recently , theophylline ( an antagonist of ventilatory depressant adenosine ) was shown to reduce CSR and oxygen-desaturation . Continuous positive airway pressure did improve CSR but not sleep and may reduce cardiac output in a subgroup of patients with heart failure . Nocturnal oxygen reduces CSR and improves exercise tolerance as well as sleep . This and its apparent safety makes oxygen an appropriate treatment for nocturnal CSR . Whether successful treatment of nocturnal CSR has any impact on the natural course of heart failure needs to be determined in further studies ."
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"As pacing impedance is inversely related to pacing current , the increase of pacing impedance additionally decreases pacing current . Whether the impedance measurement at implantation predicts the outcome during follow-up , was studied in 87 patients who received the VDD-single lead UniPass 425 connected to the pacemaker Unity ( Sulzer Intermedics ) . The impedance changes between implantation and 6 months follow-up were assessed for each patient . Similar impedance values were defined , if the two measurements were within a range = -100 to +100 . Six-months impedance was lower or higher compared to implantation , if the difference exceeded > -100 or > +100 . At implantation , impedance was 535 +/- 98 ( range : 333-811 ) and significantly increased to 604 +/- 160 ( range : 361-1150 ) after 6 months . Mean difference between the two measurements was 69 +/- 162 ( range : -336 bis +560 ) . Similar impedance had 43 ( implantation : 527 +/- 75 , 6 months : 531 +/- 87 ) , lower values 11 ( implantation : 660 +/- 83 , 6 months : 494 +/- 73 ) and higher values 33 patients ( implantation : 503 +/- 99 , 6 months : 735 +/- 168 ) . Compared to the patients with similar impedance patients with lower impedance had a significantly higher impedance values at implantation . Conclusions : Pacing impedance increased significantly within 6 months after implantation . Pacing impedance changed > 100 in 51% of the patients . The long-term follow-up of pacing impedance can be predicted generally , but not for the individual patient ."
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"The influence of a coronary dissection on long-term outcome after PTCA has been controversely discussed in the past . Whereas diverse experimental studies have shown a positive relation between dissection and the incidence of restenosis , clinical trials could not document an influence of dissection on long-term outcome . However , most of the trials did not distinguish between the different morphologic configuration of the vascular dissection . Thus , the aim of the present study was to determine the influence of dissections on restenosis in regard to their amount and morphologic configuration . The prognostic importance of the National Heart , Lung , and Blood Institute classification on dissection as well as the importance of an additional classification of angiographic complications after PTCA were investigated to determine possible pathophysiologic mechanisms of the restenosis process . The study included 141 consecutive patients with 143 stable dissections after PTCA . A follow-up study was performed 13 months in mean after successful PTCA , which included clinical , symptomatic , and functional aspects of patients . In this patient population , type C dissections ( Dorros et al. ) showed a relevantly increased risk of a clinical adverse event ( 41.0% ) , whereas patients with a type A dissection had only a small risk of an adverse event ( 10.0% ) over the investigation period . Type B dissections revealed an intermediate risk ( 18.0% ) , and type D dissections showed a risk of 33.3% of an adverse event , which was lower than that observed for type C dissections . The AC-classification of the postinterventional coronary morphology was a stronger predicter of an adverse outcome after PTCA ( p = 0.0003 ) than was the Dorros-classification ( p = 0.0056 ) . Conclusions : The grade of a coronary dissection was highly , positively related to an ischemic event after PTCA using both the Dorros and the AC-classification ( p = 0.0056/p = 0.0003 ) . In regard to the special association of the AC-class with the amount of vascular injury , we conclude that the amount and configuration of coronary dissection correlates with the long-term outcome after PTCA ."
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"Some patients with nonischemic heart failure show inflammatory changes in the myocardium which are thought to be of causal or pathogenetic relevance for the heart failure . The intercellular adhesion molecule-1 ( ICAM-1) and the vascular adhesion molecule-1 ( VCAM-1) are membrane proteins with receptor function from the immunoglobulin superfamily which mediate the vascular adhesion and transmigration of leucocytes into the tissue and undergo increased expression in chronic immunological-inflammatory processes . In addition to membrane-bound adhesion molecules , soluble forms can be detected in serum . In the present study we investigated the occurrence and the significance of circulating ICAM-1 and VCAM-1 in 71 patients with non-ischemic heart failure ( 47 M/24 F , mean age 55 +/- 11 years ) Methods : Serum concentrations of cICAM-1 and cVCAM-1 were analyzed using ELISA-Kits . The severity of heart failure was assessed in accordance to the NYHA-classification and to hemodynamic parameters ( mean pulmonary pressure , left ventricular ejection fraction ) . Inflammatory heart disease was assessed histologically and immunhistologically ( T-lymphocytes > 7.0/mm2, increased expression of the histocompatibility antigens of class I and II ) in right ventricular endomyocardial biopsies . 16 healthy , agematched patients ( 8 M/8 F , mean age : 55 +/- 6 years , mean ejection fraction 76 +/- 3% ) without signs of inflammation in the myocardium ( mean T-lymphocytes 3.5 cells/mm2, low expression of HLA-class I and II ) served as controls . Results : The mean serum concentrations of circulating ICAM-1 and V-CAM1 ( cICAM-1,cVCAM-1) were higher in patients with non-ischemic heart failure ( 372 +/- 107 ng/ml and 949 +/- 439 ng/ml ) than controls ( 264 +/- 37 and 710 +/- 164 ng/ml ) ( p 0.05 ) . The mean concentrations of both adhesion molecules varied as a function of the mean pulmonary pressure and the left ventircular ejection fracture ( for cICAM-1: Pearson's r : 0.24 and -0 . 33 , p 0.05 ; for cVCAM-1: Pearson's r : 0.28 and -0 . 26 , p 0.05 ) In 38% ( n = 16 ) of patients with elevated concentrations of cICAM-1 ( > = 337 ng/ml ) and 41% ( n = 7 ) of those with elevated serum levels of cVCAM-1 ( > = 1038 ng/ml ) , the myocaridal biopsies showed increased lymphocytic infiltration between 7 and 22 T-lymphocytes/mm2 and an enhanced expression of the MHC antigens of class I/II as sign of an activated inflammatory process in the myocardium . All patients with more than 9.3 T-lymphocytes/mm2 in the myocardium ( n = 7 ) had higher serum levels of cICAM-1 ( 447 +/- 146 ng/ml , p 0.05 compared to controls ) and of cVCAM-1 ( 1577 +/- 688 ng/ml , p 0.001 ) . Both adhesion molecules correlated significantly with the mean number of T-lymphocytes in the myocardium ( Pearson's r : 0.31-0.37, p 0.05 ) . Summary and conclusion : The present study shows that the elevated levels of cICAM-1 and cVCAM-1 are often found in the serum of patients with non-ischemic heart failure . These raised serum levels correlate with inflammatory infiltrates in the myocardial tissue and with the clinical and hemodynamic signs of heart failure , thus , confirming a connection between heart failure and inflammatory changes in the myocardium ."
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ZfuerKardiologie.80870105.eng.abstr | ZfuerKardiologie.80870105.eng.abstr | [
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}
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"Heart rate variability ( HrV ) has been established as an important risk parameter in patients surviving myocardial infaction . Recently , analysis of HRV - particularly of the standard deviation of NN intervals ( SDNN ) - from short term ECG recordings has been proposed as an alternative to that from 24 hour Holter monitorings . The present study is the first to compare SDNN from short term recordings to that from 24 hour recordings with respect to risk stratification after myocardial infarction . In 60 patients at the time of discharge from the hospital , SDNN from Holter monitoring averaged 93 +/- 30 ms compared to 39 +/- 18 determined from short term recordings . Patients with a prospectively defined clinical endpoint differed significantly from those with an uneventful course with respect to age , left ventricular ejection fraction , mean RR interval from Holter recording ( 753 +/- 153 vs 921 +/- 132 ms ) , mean RR from short term recording ( 783 +/- 153 vs 914 +/- 163 ms ) , and SDNN from the Holter recording ( 71 +/- 27 vs 97 +/- 29 ms ; p 0.001 ) However , patients with and without clinical event could not be seperated by means of SDNN measured from short term ECG registrations ( 35 +/- 22 vs 41 +/- 19 ms ; P = NS ) . Thus , SDNN measured from short term ECG recordings does not seem to be useful regarding risk stratification of patients after myocardial infarction . Analysis of HRV should be performed using 24 hour Holter monitoring , which provides more information and reflects circadian fluctuation in autonomic tone ."
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"Problem : Atrial fibrillation is associated with significant morbidity and mortality . The increased risk of thromboembolism makes constant anticoagulation necessary , while the absence of atrial contraction worsens the hemodynamic situation . In this study we examined the results of simultaneous surgical treatment of chronic atrial fibrillation through \" Cox/Maze-III-procedure \" while mitral valve surgery and atrial septal closure . Patients and methods : Between February 1995 and July 1997 sixteen patients aged 50 to 72 y ( mean : 59 y ) underwent open heart surgery ( mitral valve replacement n = 10 , reconstruction n = 3 ; mitral valve reconstruction and atrial septal closure n = 1 , atrial septal closure n = 1 , thrombectomy from left atrium n = 1 ) and \" Cox/Maze-III-procedure \" in one session . Preoperative duration of chronic atrial fibrillation was between 2 and 12 y ( nean : 5.0 y ) . The patients were examined preoperatively and postoperatively 3 , 6 , 12 , and 24 month after surgery using the following methods : transesophageal echocardiography ( TEE ) , ECG , long-term ECG , and stress ergometry . Results : During the early postoperative period ( 7 d ) all of the patients were in sinus rhythm or atrial pacing ; eight patients intermittently had atrial fibrillation . In the follow-up period from 1 to 27 months ( mean 14 months ) , 15 patients have a stable sinus rhythm ( 94% ) . One patient , preoperatively known to have intermittent tachybradycardia , received a pacemaker 2 months postoperatively for sinus bradycardia . Postoperatively in TEE patients showed left atrial contraction with improvement of hemodynamic situation . Stress ergometry 6 months postoperatively showed lower increasements of heart rate , at 12 months normal frequency . Conclusion : The surgical treatment of chronic atrial fibrillation through \" Cox/Maze-III-procedure \" can be performed combined with other heart operations without increased perioperative or postoperative risks . The Maze-procedure is an effective surgical therapy of chronic atrial fibrillation . Postoperatively there is an improvement of the hemodynamic situation of sinus rhythm and synchrone atrioventricular contraction ."
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"Adenosine is generally considered to be safe and effective in treatment of orthodromic circus movement tachycardia in patients with accessory pathways . We present a case with reproducible induction of atrial fibrillation after initial successful termination of orthodromic circus movement tachycardia using a left sided accessory pathway by adenosine . With the induction of atrial fibrillation sudden onset of preexcitation with subsequent rapid ventricular response was accompanied by moderate hemodynamic compromise . Before and after successful ablation of the accessory pathway , adenosine was not able to induce atrial fibrillation when applied during sinus rhythm . Adenosine induced atrial fibrillation is due to shortening of the atrial action potential duration and atrial refractoriness , whereas shortening of the antegrade refractory period in the accessory pathway leads to rapid conduction to the ventricles . Thus , care should be taken using adenosine in patients with orthodromic circus movement tachycardias and evidence of antegrade preexcitation during sinus rhythm ."
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"Ten young patients with a median age of 10.5 ( range : 6 to 31 ) years suffering from the permanent form of junctional reciprocating tachycardia ( PJRT ; n=7) and ectopic atrial tachycardia ( AET ; n=3) were treated by transcatheter radiofrequency current application . Indications for interventional therapy were failure of medical therapy after a median of 3 antiarrhythmic drugs in all patients , syncope in 1 , and impaired left ventricular function in 4 patients ( PJRT n=3, AET n=1). The intervention was primarily successful in all patients . Median fluoroscopy time during the ablation procedure was 17 ( 10 to 70 ) min . A median of 3 ( 1 to 14 ) radiofrequency ( 500 kHz ) pulses was delivered at a target temperature of 70 °C or with 30 W for 30 s. One patient had a recurrence of PJRT and was successfully treated by a repeat ablation . After a median follow-up of 14 ( 2 to 61 ) months , all patients are free of drug treatment and in normal sinus rhythm . Left ventricular function has normalized in patients with follow-up > 6 months . Radiofrequency catheter ablation was a safe and effective therapy in young patients with drug-resistant forms of chronic-permanent supraventricular tachycardia ."
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"Background . Electrophysiological abnormalities during ischaemia and increased heart rate may influence the detection of ventricular late potentials in the surface electrocardiogram . Whether the analysis of functional changes adds information to the risk stratification of patients prone to ventricular tachycardia is unclear . Methods . We therefore retrospectively investigated 100 selected patients ( 25 with documented , sustained ventricular tachycardia ( 230/min) ( = VT group ) , 25 resuscitated from ventricular fibrillation ( VF group ) and 50 without ventricular arrhythmias ( VT/VF group ) ) in the chronic phase after myocardial infarction . Late potential analysis was performed at rest , during atrial pacing at a rate of 100/min and 120/min ( n=60), during and after occlusion of the coronary artery for coronary angioplasty ( PTCA ) ( n=70), and immediately after maximum exercise using selective signal averaging . Results . At rest in 72% of patients in the VT group , in 32% of the VF group , and in 6% of the VT/VF group late potentials could be found . During atrial pacing in 80% of patients in the VT group , in 72% of the VF group , and in 10% of the patients in the VT/VF group and during ischaemia because of occluded coronary artery in 86% of patients in the VT group , 70% of the VF group , and in 20% of the patients of the VT/VF group late potentials were present . Immediately after maximum exercise which let to both ischaemia and increased heart rate , late potentials were detectable in 92% of patients in the VT group , 80% of the VF group , and in 14% of patients in the VT/VF group . Similar results could be achieved by using the Holter-ECG after exact correction of recorder tape speed variations . 62% of patients with only by ischaemia , increased heart rate or exercise provokable late potentials and all patients with preexistent not by PTCA extinguished late potentials developed recurrent ventricular tachycardias during the one year follow-up period . Patients without late potentials ( n=50) and patients with preexistent by PTCA extinguished late potentials ( n=11) had no recurrent ventricular tachycardias . Cycle length of recurrent and clinical tachycardia in patients with preexistent not by PTCA extinguished late potentials ( n=18) were significantly longer than in patients with only provokable late potentials ( n=21). Conclusions . Analysis of functional changes of ventricular late potentials with exercise or in Holter ECG recordings promises considerable improvement of postinfarction risk stratification especially in patients prone to ventricular fibrillation ."
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ZfuerKardiologie.80870478.eng.abstr | ZfuerKardiologie.80870478.eng.abstr | [
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"Electron beam CT , which does not require mechanical movement of an X-ray tube , has a temporal resolution which exceeds that of conventional computed tomography by a factor of about ten . Axial images of the heart can be acquired within 50 to 100 ms with a spatial resolution below 0.5 mm2 and permit precise visualization of all cardiac structures . ECG-triggered acquisition of 30 to 40 axial images ( 3 mm slice thickness ) in a short inspiratory breathhold allows one to sample a 3-dimensional volume data set which covers the complete heart . Overall acquisition times are approximately 30 to 50 seconds . While electron beam CT in general permits a complete cardiac investigation , including morphology , function , and perfusion , the method's most important application is non-invasive imaging of the coronary arteries . Without contrast enhancement , coronary calcification by EBCT currently constitutes the most sensitive non-invasive marker for the presence even of very early forms of coronary atherosclerosis . Intraveneous injection of contrast agent during image acquisition additionally permits the selective visualization of the coronary artery lumen and detection of significant stenoses . This method is especially well suited in the follow-up of coronary interventions and coronary bypass grafting . Breathhold and movement artifacts , superposition of coronary arteries and veins , as well as severe coronary calcifications currently constitute the method's main problems . In spite of these limitations , electron beam CT has been shown to permit clinically valuable non-invasive investigations of the coronary arteries , which may be further refined as technology progresses ."
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"Reports from several countries indicate that women are disadvantaged in the treatment of coronary heart disease . The aim of the present review is to examine - on the basis of current population-based data ( National Health Survey [ NHS ] , hospital diagnosis registry , mortality rates ) - whether in the Federal Republic of Germany more recently a change in favor of women could be established . According to NHS results prevalence rates of hypertension , overweight , and smoking in men exceed those of women , whereas hypercholesterolaemia is equally prevalent in both sexes . From 1984/85 to 1990/91 the NHS revealed a marked decline of smoking in men , and a parallel increase in women . The prevalence of chest pain shows no gender difference , but chest pain in men is more predictive for coronary artery disease . In acute myocardial infarction [ AMI ] thrombolysis and PTCA are applied with equal frequency ; the average length of stay in hospital is greater for women . Coronary bypass surgery and rehabilitation in hospitals are less frequently applied in women . Little is known about gender differences in psychosocial adaptation after AMI . AMI mortality rates in all age groups are lower in women , and their mean age of death is higher . However , the decline of AMI mortality since 1980 was less pronounced in women compared to men ."
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"This prospective study was designed to compare quality of life , life satisfaction , and subjective ratings of health before and at variable time intervals after heart transplantation ( HTx ) . 175 patients were included between February 1994 and December 1997. Every six months before and 1,5 , 3 , 6 , and 12 months after HTx , they received the following standardized and validated questionnaires : German SF 36 , heart failure and specific transplant symptom list , global quality of life assessment , Munich life quality dimension list , expected/experienced life changes after HTx , good command of the German language , and a minimum age of 18 years . During data evaluation , median ( Me ) , mean ( M ) , and standard deviation ( SD ) were created from individual parameters . Quality of life was rated as very poor by 84% of patients with congestive heart failure . Only 6 weeks after HTx , 74% rated their quality of life as significantly better . Before HTx 80% were very unsatisfied about their health status and 87% about physical performance . Six weeks after HTx , this parameter improved significantly and 76% were very satisfied about their health status and 50% about physical performance . While somatic changes expected before HTx corresponded well with experienced ones , psychological improvements were smaller than expected , but one year after HTx they were significant ( before : M=3.66; SD=0.9; Range ( R ) =3 . 78 vs 12 months postop : M=4.61; SD=0.6; R=2.67; p 0.05 ) . Quality of life correlated before HTx best with subjectively rated health ( r=0.61, p 0.01 ) and 6 months after with satisfaction about health status ( r=0.76, p 0.01 ) . Only in 25% were expected improvements fulfilled regarding sexual activity/satisfaction , professional situation , and recreational activities . 90% of posttransplant patients reported physical compaints , most by effects of immunsuppression , but were coping well . The study shows that already 6 weeks after successful HTx all quality of life parameters improved significantly . Despite some unfulfilled expectations and complaints , the postoperative life situation of HTx patients appeared significantly improved ."
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"Patients with a dual chamber pacemaker often suffer from chronic heart failure . The influence of the upper tracking rate on cardiac performance in patients with and without congestive heart failure is unknown . Therefore , twenty patients with a dual chamber pacemaker implanted for high degree AV block were randomly programmed to upper tracking rates of 110 , 120 , and 130 bpm . Oxygen uptake ( VO2), work capacity , and heart rate were determined at the anaerobic threshold ( AT ) and at peak exercise using spiroergometry every 4 weeks . Nine patients ( 71 +/- 12 years ) had evidence for advanced heart failure ( Weber C/D , group I ) ; 11 patients ( 60 +/- 6 years ) had no or only mild heart failure ( Weber A/B , group II ) . Patients in group II achieved a higher mean VO2-AT at 130 bpm ( 17.3 +/- 3.9 ml/min/kg ) than at 110 bpm ( 13.7 +/- 4.0 ml/min/kg ; p=0.001). Maximum oxygen uptake and work capacity at the anaerobic threshold were also significantly higher in group II with an upper tracking rate of 130 bpm than at 110 bpm ( 20.5 +/- 4.5 ml/min/kg vs. 18.2 +/- 5.3 ml/min/kg , p=0.031, and 98 +/- 29 W vs. 86 +/- 27 W , p=0.04). In group I , mean oxygen uptake at the anaerobic threshold ( VO2-AT) was higher at 110 bpm ( 11.1 +/- 4.0 ml/min/kg ) than at 130 bpm , although of only borderline statistical significance ( 9.2 +/- 2.6 ml/min/kg ; p=0.052). In group I a higher upper rate decreased VO2-AT by 24% , whereas in group II the higher upper rate improved aerobic capacity by 26% . Stressechocardiography ( VVI pacing with a maximum tracking rate of 130 bpm ) revealed new wall motion abnormalities in all patients of group II . Wall motion score index increased from 1.20 +/- 0.24 at rest to 1.54 +/- 0.28 under stress ( p 0.001 ) in group I , but not in group II ( 1.00 +/- 0 vs. 1.06 +/- 0.19 ; p=n.s.). Thus , carriers of dual chamber pacemakers with no or mild heart failure ( Weber A/B ) benefit from higher programmed upper rates . In contrast , patients with more advanced heart failure ( Weber C/D ) improve aerobic capacity with lower programmed upper rates . This may be caused by exercise-induced ischemia in group I as indicated by stressechocardiography ."
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"The purpose of our study was to determine the prevalence of sleep related breathing disorders ( SRBD ) in patients with an implantable cardioverter-defibrillator ( ICD ) and to evaluate prospectively the possible influence of SRBD on arrhythmia recurrence and circadian arrhythmia variation as well as on cardiac mortality during long-term follow-up . Forty consecutive ICD recipients with cardiac disease and a documented history of spontaneous , life-threatening , ventricular tachyarrhythmias underwent fullnight polysomnography and were followed for 2 years . In 16 of 40 patients ( 40% ) , SRBD were diagnosed ( Apnea/Hypopnea Index ( AHI ) > 10 ) ; in 9 of these 16 patients ( 56% ) central sleep apneas ( CSA ) occurred ( in 8 of these 9 patients in combination with Cheyne-Stokes respiration ) . Seven of the 16 patients with SRBD ( 44% ) revealed obstructive sleep apneas ( OSA ) . AHI was 32 +/- 15 ( 12-60 ) in patients with CSA and 32 +/- 27 ( 11-86 ) in patients with OSA . Patients with and without SRBD were comparable concerning left ventricular ejection fraction , NYHA classification , cardiac disease , ICD indication , and concomitant medication . ICD registered ventricular tachyarrhythmias occurred in 10 of 24 patients ( 42% ) without SRBD , in 4 of 9 patients ( 44% ) with CSA , and in 3 of 7 patients ( 44% ) with OSA . The numbers and circadian variation of episodes registered during follow-up in patients without SRBD , with OSA or CSA were comparable ( 14 +/- 25 , median 4 vs 15 +/- 15 , median 7 vs 4 +/- 5 , median 2.5 ) . The 2-year cardiac mortality was highest in patients with CSA ( 4/9 ( 44% ) vs 0/7 patients ( 0% ) with OSA vs 3/24 patients ( 12.5% ) without SRBD . Thus , the prevalence of SRBD in patients with chronic heart failure and a history of malignant ventricular tachyarrhythmias is high ( 40% ) and the occurrence of CSA seems to be predictive for cardiac mortality in these patients . An influence of moderate SRBD on arrhythmia recurrence and circadian variation of spontaneous sustained tachyarrhythmic events could not be demonstrated ."
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"Cor triatriatum dexter is a rare congenital malformation in which a membrane divides the right atrium into two chambers . The membrane represents a persistence of the right sinus venosus valve ( RSV ) . Normally the RSV regresses between the 9th and 15th week of gestation , as the cephalic portion forms the crista terminalis and the caudal portion develops into the Eustachian and Thebesian valve . Any failure in the regression process may result in remnants of RSV as a simple muscle bar , a Chiari-network or a fenestrated or unfenestrated membrane ( cor triatriatum dexter ) . We describe a patient with cor triatriatum dexter in whom diagnosis was made several years after successful valvulotomy procedure for severe congenital valvular pulmonary stenosis ."
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"Spontaneous coronary artery dissection is a rare cause of ischemic heart disease . Incidence , etiology and optimal treatment are ill-defined . Between July 1995 and December 1997 , we prospectively identified 42 patients ( 36 men , six women , mean age 59 +/- 12 years ) with spontaneous coronary artery dissection among 3803 consecutive angiographic examinations in which the diagnosis of coronary artery disease was established for the first time ( incidence 1.1% ) . In comparison to the remaining study population with stable angina pectoris ( 8 cases of spontaneous coronary artery dissection among 2852 patients ; incidence : 0.3% ) , the incidence of spontaneous coronary artery dissection was significantly higher in the patient subgroups with acute myocardial infarction ( 13/450 ; 2.9% ) and with unstable angina pectoris or postinfarction angina ( 21/501 ; 4.2% ) . Dissection was most frequently located in the left anterior descending coronary artery ( 19 cases ) , followed by the right coronary artery ( 15 cases ) and the left circumflex coronary artery ( 8 cases ) . Because of an ambiguous angiographic lesion appearance intravascular ultrasound imaging was performed in 13 patients to confirm the diagnosis . The presumed etiology of spontaneous coronary artery dissection was atherosclerotic plaque rupture in 35 cases , heavy physical exercise in four cases and hormonal influences related to pregnancy and contraception in one case . In two cases , no obvious risk factor could be identified . Therapy consisted of intracoronary stenting in 24 patients ( including ten patients with acute myocardial infarction ) , coronary artery bypass grafting ( CABG ) in 8 patients and balloon angioplasty ( PTCA ) in seven patients . Three patients were treated conservatively ."
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ZfuerKardiologie.8087s020.eng.abstr | ZfuerKardiologie.8087s020.eng.abstr | [
{
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"The morphological correlate of acute coronary syndromes is a ruptured plaque with intraluminal thrombus formation . Furthermore , inflammation , coagulation , fibrinolysis , complement system , and corpuscular elements of the blood are activated . Certain markers as CRP , fibrinogen , plasminogen activator inhibitor or tissue-type plasminogen activator antigen are reported to have a prognostic impact to identify patients at risk . Morphological data provide evidence for the impact of inflammatory mechanisms for the rupture of an unstable plaque and an association of several infections with coronary syndromes was reported . To date it is not known to what extent the various pathomechanisms influence the rupture of a plaque with the risk of consecutive formation of an occlusive thrombus . It can be assumed that a plaque rupture without critical occlusion of the vessel occurs frequently without symptoms , thus making the evaluation of causal relationships clinically difficult . Future studies should investigate the complex interactions of the different systems by determination of markers of activation in parallel during the acute phase of the disease . Furthermore , experiments should be designed to examine the relative impact of single factors for the pathophysiological process of plaque rupture in acute coronary syndromes including the meaning of infections agents ."
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ZfuerKardiologie.8087s041.eng.abstr | ZfuerKardiologie.8087s041.eng.abstr | [
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"During moderate prolonged myocardial ischemia , the myocardium is dysfunctional but can remain viable . In such ischemic and dysfunctional myocardium , contractile function is reduced in proportion to the reduction in regional myocardial blood flow , i.e. , a state of \" perfusion-contraction matching \" exists . The metabolic status of such myocardium improves over the first few hours , as myocardial lactate production is attenuated and creatine phosphate , after an initial reduction , returns to control values . Ischemic myocardium , characterized by perfusion-contraction matching , metabolic recovery and lack of necrosis , has been termed \" short-term hibernating myocardium \" . \" Short-term hibernating \" myocardium can respond to an inotropic stimulation with increased contractile function , however , at the expense of a renewed worsening of the metabolic status . A role for endogenous adenosine in the development of hibernation has been excluded , since neither contractile function , metabolic parameters , nor viability are altered by increased catabolism of endogenous adenosine by infusion of adenosine deaminase . Also activation of ATP-dependent potassium channels is not responsible for \" short-term hibernation \" . \" Short-term hibernating \" myocardium has , however , reduced calcium responsiveness ."
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"Background : An abnormal coronary flow reserve represents an early marker of impaired blood flow regulation in the natural history of coronary atherosclerosis under the impact of risk factors such as hypercholesterolemia . Our clinical investigation was aimed at assessing noninvasively the integrative coronary flow response to dipyridamole stress in 18 consecutive patients with microvascular angina , only moderately elevated LDL-cholesterol levels ( 168 +/- 33 mg/dl ) , and reduced vasodilator capacity despite normal ( n=9) or slightly abnormal ( n=9) coronary arteriograms ( minimal disease with luminal irregularities and/or diameter reduction =30% ) before and after 6-month lipid-lowering therapy ( simvastatin ) . Methods : Regional and averaged myocardial blood flow were measured at rest and after dipyridamole induced vasodilation ( 0.56 mg/kg ) using dynamic positron emission tomography ( PET ) and N-13 ammonia as flow tracer related to a 3-compartment kinetic model . Baseline data ( mean +/- SD ) : 13 males , 5 females ; mean age : 56 +/- 8 years ; basal coronary flow : 90 +/- 22 ml/minx100 g ; after lipid intervention : 93 +/- 18 ml/minx100 g ( n.s. ) . Total cholesterol decreased to 170 +/- 36 mg/dl ( p 0.001 ) ; mean LDL level : 97 +/- 26 mg/dl ( p 0.001 ) . Coronary dilator capacity increased , assessed in terms of minimal coronary resistance : 0.38 +/- 0.08 vs 0.49 +/- 0.09 units at baseline ( p 0.01 ) , myocardial blood flow under dipyridamole : 232 +/- 43 vs 186 +/- 37 ml/minx100 g at baseline ( p 0.01 ) , and instantaneous flow ratio : 2.6 +/- 0.7 vs 2.2 +/- 0.6 ( p=0.06). Concomitantly , a considerable regression of angina was noticed in the majority of patients . Conclusions : An improvement of the non-invasively determined integrative dipyridamole induced coronary vasodilator capacity may be achieved after 6 months by intensive lipid lowering at a very early stage of coronary atherosclerosis . Consequently , aggressive cholesterol-lowering therapy represents an antiischemic and antianginal approach suggesting , at least in part , functional reversal and probably prevention of further disease progression ."
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] |
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